Dietary fat has no effect on insulin levels. Insulin is responsible for the storage of fat in the body. Carbohydrates stimulate the secretion of insulin. Sweetness is carbohydrates. Proteins (proteins) also have a similar effect, but fat has zero value in this matter.
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They are getting fat from their lazy ass. Dmitry told the rest perfectly. For biochemistry is our everything! I will only add that insulin "drives" glucose into almost all tissues (except for the nervous one). And where the need for it is high, it goes there first.
The question is very interesting! Human food consists of proteins, fats and carbohydrates. Everyone knows that. Proteins are good for you by default. The author of the question asks what is more harmful (in order to get fat): fats or carbohydrates (sweet is sugar, these are ugdevods). The answer is that carbohydrates are actually more harmful in terms of weight gain. There is a lot of information about this. Fats hardly accumulate directly in the body. The excess is removed. What we accumulate is not the fat that we have consumed with food, but our own human fat, into which excess carbohydrates are processed.
It's often about the quality of food. There are much more serious reasons for being overweight: genetics and epigenetics.
Probably, each of us has a friend who devours sweets and burgers for four, but remains thin or even skinny. Conversely, who has not met people who claim that even water is immediately deposited on their waists? "Propensity to be overweight" is probably the oldest conditionally acceptable justification for obesity, but only in recent years have scientists finally begun to understand what it is at the genetic level.
The first and most important thing that emerged during research: a genetic predisposition to obesity actually exists (although many are still convinced that it is only promiscuity and laziness).
Today, scientists know more than 60 sites in the genome, certain variants of which increase the risk of "recruiting unnecessary ". Moreover, the same result - an increased tendency of the body to store unused calories in the form of fat - can be determined by completely different genetic characteristics. For example, obesity can be monogenic and polygenic, that is, caused by one or more genes. Monogenic obesity is quite rare and is associated with a "breakdown" in one of the genes encoding proteins of a special body system that regulates how much energy should be saved for the future, that is, converted into fat.
Polygenic obesity occurs when several genes are not working correctly at once. Disruption of the activity of each of them may not give a noticeable effect, but, adding up, they significantly increase the risk of acquiring Rubens' forms. For example, a geneticist-epidemiologist from the University of Michigan Elizabeth Speliots cites the following data: a person whose DNA contains 104 “wrong” variants of genes associated with obesity is, on average, 11 kilograms heavier than the carrier of 78 such variants.
What concerns epigenetics, sometimes scientists do not know why certain superstructures suddenly appear on DNA, but sometimes it is possible to trace the exact cause. And in the case of obesity, adipose tissue itself is very often the trigger. In December 2015, researchers from Denmark and Sweden demonstrated this wonderfully by comparing sperm DNA from lean and overweight men. In total, they found more than nine thousand changes in the "obese" sperm, and 274 of them were in the genes that are responsible for appetite and eating behavior.
But the most striking thing happened after some of the men with excess weight decided on liposuction. A week after the operation, 1509 epigenetic changes were formed in the DNA of their sperm, and a year after the procedure, this number increased to 3910. Although the epigenetic profile of the germ cells of artificially thin men differed from naturally slender sperm, this difference could not be compared with the difference between sperm thin and fat. As the authors note, their work indirectly proves that fat fathers through the sex cells of thegive the children approximately the following instructions: "Son / daughter, these are fat times, eat as much as possible - when there will still be this." Evolution has provided us with an excellent mechanism for quickly responding to sudden abundance, but how did she know that it would become a problem for Homo sapiens.
The mother's diet also changes the epigenetic profile of children in early pregnancy, and these changes are completely logical and correct if you are a Cro-Magnon living from hand to mouth. For example, if a mother-to-be neglects vegetables, fruits and other sources of carbohydrates, the body decides that there is a famine on the planet, and marks appear on the DNA of the children that are being carried, making their bodies more spare. In modern conditions, this means those very extra centimeters "from everything." /
On a sweet diet, you really run the risk of gaining more weight than if you ate fatty foods with the exception of sweets. Simple carbohydrates, in particular glucose, give the food a sweet taste. If the level of glucose in the blood rises, then the production of insulin increases, under the influence of which the accumulation of fats in adipocytes occurs. In addition, an excess of carbohydrates, after going through a series of biochemical changes, turns into fats.
Get fat from an excess of calories. That is, even fruits and cereals can be the cause of excess weight if you eat too much of them. Another thing is that it is more difficult to exceed the daily colloration with healthy food. So, answering your question, I can say: they get fat from both sweets and fatty and even healthy foods.